Contractile reserve but not tension is reduced in monocrotaline-induced right ventricular hypertrophy

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Contractile reserve but not tension is reduced in monocrotaline-induced right ventricular hypertrophy.

The objective of this study was to evaluate the role of right ventricular hypertrophy on developed tension (F(dev)) and contractile reserve of rat papillary muscle by using a model of monocrotaline (Mct)-induced pulmonary hypertension. Calcium handling and the influence of bicarbonate (HCO(3)(-)) were also addressed with the use of two different buffers (HCO(3)(-) and HEPES). Wistar rats were i...

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beta-Receptors and contractile reserve in left ventricular hypertrophy.

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Preservation of diastolic function in monocrotaline-induced right ventricular hypertrophy in rats.

During ischemic heart diseases and when heart failure progresses depletion of myocardial energy stores occurs. D-Ribose (R) has been shown to improve cardiac function and energy status after ischemia. Folic acid (FA) is an essential cofactor in the formation of adenine nucleotides. Therefore, we assessed whether chronic R-FA administration during the development of hypertrophy resulted in an im...

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Acceleration of Ca2+ waves in monocrotaline-induced right ventricular hypertrophy in the rat.

BACKGROUND Triggered arrhythmias arise from delayed afterdepolarizations (DADs), with Ca(2+) waves playing an important role in their formation. In ventricular hypertrophy, however, it remains unclear how Ca(2+) waves change their propagation features and affect arrhythmogenesis. We addressed this important issue in a rat model of hypertrophy. METHODS AND RESULTS Rats were given a subcutaneou...

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Preservation of diastolic function in monocrotaline-induced right ventricular hypertrophy in the rat

During ischemic heart diseases and when heart failure progresses depletion of myocardial energy stores occurs. D-ribose (R) has been shown to improve cardiac function and energy status after ischemia. Folic acid (Fa) is an essential co-factor in the formation of adenine nucleotides. Therefore, we assessed whether chronic R/Fa administration during the development of hypertrophy resulted in an i...

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ژورنال

عنوان ژورنال: American Journal of Physiology-Heart and Circulatory Physiology

سال: 2004

ISSN: 0363-6135,1522-1539

DOI: 10.1152/ajpheart.00536.2002